Scalp peeling with 20% glycolic acid: GREAT RESULTS...

*bump*
Anyone have much luck with scalp peels? Including any results from apple cider vinegar. I dont use any hair products anymore but considering starting the nopoo method (with baking soda/ACV). I tried using ACV for the first time a few weeks ago and some ran down my face and into my eye - it fucking caned! Scalp felt great after though.

Hello,

Does this 20% Glycolic acid can do the stuff ?

http://www.amazon.com/Skin-Obsession-Glycolic-Rosacea-Chemical/dp/B0018QSLJM/ref=sr_1_1?s=beauty&ie=UTF8&qid=1455031576&sr=1-1&keywords=Glycolic+acid+20

The goal is to use it once a week, yes ? But i have difficulties to understand the goal...
I already use dmso peels in the past whitout results...

The original post is missing the full explanation .

This is it.
http://www.longecity.org/forum/topic/54726-pathogenesis-of-male-pattern-baldness-the-solutions/

"The pathogenesis of male-pattern-baldness (MPB) is the same as acne. The first step in acne is a condition called follicular-hyperkeratinization or hyperkeratosis. Simply put, this means excess skin production inside and around the hair-follicle. The next step is caused by excess sebum production, followed by P.Acnes infestation of the pilosebaceous-follicle, with subsequent inflammation. So to summarize, MPB, just like acne, is caused by four main factors: (1) follicular-hyperkeratinization, (2) excess sebum production, (3) P.Acnes, and (4) inflammation [1].

Free-IGF-1 (insulin-like-growth-factor-1) stimulates the proliferation of keratinocytes (skin cells below the scalp). As the keratinocytes mature, they migrate to the top of the scalp and become what is known as corneocytes. If all goes well, the corneocytes are supposed to be shed off the scalp in a process called apoptosis. The shedding of corneocytes or the apoptosis of corneocytes, is under the influence of IGFBP-3 (insulin-like-growth-factor-binding-protein-3) and endogenous retinoids. In men with male-pattern-baldness, they have excess free-IGF-1 and not enough IGFBP-3 [2]. The result of this is hyperkeratinization; too much skin production, and not enough skin exfoliation. This causes the hair-follicle to be blocked by excess skin. Free-IGF-1 is required for keratinocyte proliferation in humans [3] and too much IGF-1 results in hyperkeratosis [4].

Free-testosterone stimulates sebum production by converting into DHT (dihydrotestosterone) within the sebaceous-glands. Both insulin and free-IGF-1 can not only stimulate the synthesis of androgens from testicular-tissue [5] [6], but high levels of both insulin and free-IGF-1 also inhibit the livers production of SHBG (sex-hormone-binding globulin) [7] [8]. SHBG is shown to be lower in men and woman with androgenic-alopecia (MPB) [9-13]. As SHBG goes up, free-testosterone goes down. As free-testosterone goes down, less is available to be converted into DHT. With less DHT comes less sebaceous-activity.

So far I have shown that a lack of IGFBP-3 (and excess free-IGF-1) causes hyperkeratinization of hair-follicles. And that low levels of SHBG promote excess sebum production. Next, after combining these two scenarios, we get P.Acnes infiltration. You see, the excess skin is now blocking not only the hair-follicle from sprouting out of the scalp, but now, also the flow or excretion of sebum. As a consequence, the sebum builds-up within the pilosebaceous-follicle. With this anaerobic-environment caused by the hyperkeratosis and with all this trapped excess sebum, the situation becomes the perfect breeding ground for a bacterium named P.Acnes, which feeds off the trapped sebum. The body is smart and the immune-system detects these nasty critters and attacks them with inflammation [14]. This explains why Nizoral-shampoo seems to be effective in controlling the symptoms of MPB [15]. The active ingredient in Nizoral-shampoo is ketoconazole, which has been shown to kill P.Acnes [16].

The solution to this cascade of events, would be to obviously lower your free-IGF-1 activity. Lowering free-IGF-1 would, (1) decrease the IGF-1:IGFBP-3 ratio, and (2) increase SHBG, thus preventing hyperkeratinization of the hair-follicles and excess sebum production. Preventing hyperkeratosis and sebum production would prevent P.acnes infestation and the subsequent inflammation that follows.

So how does one lower free-IGF-1 levels? Studies are consistent in showing that a vegan-diet causes both a decrease in IGF-1 and an increase in SHBG [17-31]. Many of these studies indicate that vegans have higher SHBG and lower IGF-1 despite the fact they are consuming high-fat diets, like eating lots of nuts or avocados. I personally believe that an extremely-low-fat vegan diet is optimal, due to the fact that dietary-fat causes insulin-resistance. Insulin-resistance causes high-insulin and IGF-1 in the blood. I also believe that fructose should be limited as much as possible. Fructose readily converts into fat in the liver, in a process called de-novo-lipogenesis and once again, fat paralyzes insulin-action, thus keeping it in the blood for extended periods of time, instead of driving glucose into the cells. Animal-protein, especially protein from dairy-products, rich in methionine, casein, and essential amino-acids seems to be the best stimulator of IGF-1. This explains why vegan-diets, low in "high-quality" protein increase SHBG and decrease IGF-1 regardless of fat content. With that stated I personally believe with all of my heart, that you would get the best results with a diet consisting only of beans, whole-grains and vegetables. Nuts and fruit (fat and sugar) should be eaten rarely.

Avoiding dairy-products is of huge importance! Nothing increases free-IGF-1 more than dairy-products; this has been consistently shown over and over again in the scientific-literature. And this should come as no surprise, considering the purpose of milk is to stimulate rapid-growth, development and maturity of the newborn. Dairy-products also contain DHT precursors [32] [33] and potent estrogens [34-36].

After over 30 years of nutritional dogma barking over and over again that "diet has nothing to do with acne". Recent research has proven beyond a doubt, that diet does affect acne and that dairy-products and high-fat, sugar-laden processed-food are the largest culprits [37-45]. And since the pathology of MPB and acne are identical, any type of diet that promotes acne has the potential to promote MPB in genetically-predisposed people.

Eating a low-fat, low-fructose, high-fiber vegan-diet high in complex-carbohydrates should stop the progression of MPB hormonally. However this is unlikely to get rid of all the follicular-hyperkeratinization that has already occurred over time. To reverse hyperkeratosis, exfoliate the scalp and regrow "lost" hair, I highly suggest using skin peels topically. Alpha-hydroxy-acids are shown to diminish corneocyte cohesion, induce exfoliation and reverse hyperkeratosis [46]. This should promote regrowth.

So to rehash or recap everything said here: a high level of free-IGF-1 (caused by eating a high-fat, high-protein, rich western-diet) causes follicular-hyperkeratinization and down-regulation of SHBG. Low SHBG correlates with the metabolic-syndrome [47-51] and promotes excess sebum production. Excess sebum is trapped, along with the hair-follicle, beneath the excess skin (produced by over-expression of IGF-1), this gives rise to P.Acnes; a bacterium that feeds off sebum and generates inflammation from the immune-system. Eating a diet consisting of beans, whole-grains and vegetables will drastically decrease IGF-1 and increase SHBG, preventing any further progression of MPB. Glycolic-acid used topically will reverse the hyperkeratosis already present and promote regrowth.

I would also like to close by saying that the effects of the complex-carbohydrate-vegan-diet can be magnified by practicing intermediate-fasting, exercise, getting plenty of sunshine exposure on the body and drinking nothing but water and organic-green-tea.
"

The reason most hair protocols fail is because there is an overgrowth/layer of skin on the scalp. Unless it is peeled away gradually, nothing can grow.

Peels are absolutely essential for hair growth. Some good news and bad news.

Gycolic acid or any acid peels don't work all that well. I tried glycolic and it did almost nothing. The best peel in the world for hair loss (that I know of) is the one from "A natural difference". 20% strength one is a miracle. The bad news is now they don't sell 20% unless you have a chemical peel license. If someone finds a way to get A Natural Difference peel at 20% strength (aka know someone or have a peel license) then let me know!


Using that one peel you literally see new hairs growing either right away or 3 days after using it.

I have been using a 10% peel from A Natural Difference since I cant get the 20% at the moment. The 20% is A LOT better than the 10%. The 10% is.. meh but the funny thing is, even at 10% strength I'm noticing it works better than many of these other peels at a higher strength. At least I do see new growth after use, even though not much.

At 10% strength I need to leave it on for a long time. I left it on overnight this week and saw some nice regrowth on my hairline within 1-2 days.

But in the end ......... if someone here could find a way to get the 20% A Natural Difference peel, people would be amazed. I have no connection with the company whatsoever and cant even get the 20% one myself right now.

Has anyone explored this further? I'm looking into the A Natural Difference brand.