Boron & Ca metabolism & hairloss

jdp710 wrote:I've taken boron supplements and borax as 20 mule laundry detergent for 2 years off and on and have a bunch of literature printed out about it.

Appears to work extremely well if magnesium deficient. I'm personally of the opinion that it may not do much if not magnesium or vitamin d, etc. deficient in regards to calcification.

Appears to work extremely well in regards to arthritis but again, I have my doubts how well it works if not magnesium etc. deficient.

Possibly a very good treatment for candida when mixed with high dose Iodine.

Appears to work extremely well to chelate flouride. May work even better using Iodine and boron to chelate flouride.

Can be applied topically and will be absorbed in your body. Just buy 20 mule borax laundry detergent and apply with a little bit of water topically or elsewhere.

Interestingly, when I started topical borax on my scalp by dunking my head in warm water mixed with a lot of borax for several minutes or applied topically or used with diluted hydrogen peroxide mixed with borax and sprayed topically I had "THE" worst shed I've ever had in my life. I still don't know to this day if it was a good shed but I occasionally revist it and can't reproduce a shed or any benefits.

Apple cider vinegar is high in boron.

Boron is helpful regarding radiation.

hope this helps

..."When examining the data from the National Health and Nutrition Examination Survey (NHANES), researchers uncovered a significant relationship between boron and prostate cancer risk. After comparing the diets of nearly 8,000 men, they found that the risk of prostate cancer for men consuming an average of 1.8 milligrams of boron was less than one-third the risk for men consuming half that amount."...

Dr. Wright admits it's a bit early to know for sure, but recent research indicates that boron (a nutrient found in vegetables and fruits) may prevent prostate cancer and autoimmune diseases (including lupus, Graves' disease, Hashimoto's disease, type-1 diabetes, vitiligo, multiple sclerosis, and more).

When examining the data from the National Health and Nutrition Examination Survey (NHANES), researchers uncovered a significant relationship between boron and prostate cancer risk. After comparing the diets of nearly 8,000 men, they found that the risk of prostate cancer for men consuming an average of 1.8 milligrams of boron was less than one-third the risk for men consuming half that amount.

Another group of researchers, from the USDA's Human Nutrition Research Center in Grand Forks, Idaho, reported that studies on animals have shown that the equivalent of 2 milligrams of boron taken daily prevents the activation of "T-helper" and "T-suppressor" cells, both of which are involved in autoimmune disease. These results were significant enough to persuade the researchers to launch a study of supplemental boron as a treatment for rheumatoid arthritis, which is often cited as an autoimmune disease.

What should you do? Well, you can take supplemental boron. It's safe (when taken in low doses -- up to 6 milligrams per day), inexpensive, and available in nearly all natural food stores. And, of course, don't forget to eat your fruits and vegetables -- they're by far the best sources of boron.

If you're not sure about boron yet, wait until you hear this: A few years ago Dr. Forrest Nielsen (at the USDA research center) showed that boron helped raise levels of sex hormones internally in women past menopause and in men of the same age group.

With all of the bad news about synthetic hormone replacement therapy, it's a relief to know that something as simple and widely available as boron might help boost your hormone levels safely and naturally.

In a double blind trial comparing 6 mg/d of boron with placebo in the treatment of arthritis, of the 10 patients on boron, five improved while only one of ten in the placebo group improved. The boron had significant benefit in severe osteoarthritis. The 6 mg of boron was in two tablets containing 25 mg of borax (sodium tetraborate decahydrate) . The experiment was carried over an eight week period. There were no side effects noted.

"Boron and Arthritis: The Results of a Double-Blind Pilot Study", Travers, Richard L. , MD, et al, Journal of Nutritional Medicine, 1990:1:127-132. (Address: Rex E. Newnham, Ph.D., Cracoe House Cottage, Cracoc Near Skipton, North Yorkshire 5023 6LB, United Kingdom)

Osteoarthritis and Boron

Six mgs/d of Boron (2 tablets of 25 mg Borax Sodium Tetraborate Decahydrate) was evaluated in 10 patients with radiographically proven osteoarthritis while 10 patients received placebos. Out of the 10 patients taking boron 5 improved while only 1 improved in the placebo group. There were no apparent side effects and the author suggests this limited trial should encourage further investigation.

"Clinical Trial — Boron on Arthritis", Travers, Richard L. , Townsend Letter For Doctors, June 1990360-362. (Address: Dr. Richard L. Travers, Department of Medicine, Royal Melbourne Hospital, Parkville 3050, Australia)

http://www.afpafitness.com/articles/ArthritisAlternatives.htm

Current research points to boron's being the equivalent of a biochemical helper in bone health, rather than a front-line soldier. But scientists also cite evidence that boron somehow prevents calcium from being excreted, plays a part in the correct balance of other minerals, and helps the body to avoid osteoporosis in the first place. It certainly seems to be central to the biochemical equation.

Boron may also have a role in another debilitating bone condition - arthritis. Arthritis is almost unknown in Israel, where levels of boron in the soil - and therefore in foods - are high. In Jamaica, however, it's the reverse situation: boron levels are low and arthritis rates are correspondingly high. That boron may have a role in the prevention or modification of arthritis is supported by research done with arthritis-susceptible rats by the U.S. Department of Agriculture. From this research, USDA scientists hypothesize that boron may modulate the immune system and protect against inflammatory disease. However, much work must be done before scientists can say that boron can help reduce the human suffering caused by arthritis.


http://www.encognitive.com/node/2124

BORON

Very little research has been done on boron and little is known about the symptoms of boron deficiency. Following we piece together a picture that indicates that boron is essential for magnesium and calcium metabolism, and is probably involved in estrogen and testosterone metabolism. There are a lot of reasons to suspect that a boron deficiency is involved in hyperthyroidism.

The following study suggests that boron works with magnesium and this may be one reason that it benefits persons with hyperthyroidism or persons with thyroid disease who are experiencing low magnesium symptoms like rapid heart rate and muscle cramping. You will note that boron both lessens the effects of a low magnesium diet but exacerbates deficiency symptoms. These seem to be the typical characteristics of when one nutrient works with another. Boron thus seems essential for magnesium metabolism and administration of boron will lower magnesium levels because it is enabling more of the magnesium to be utilized.
Another interesting observation in this study is that fructose mimics a magnesium deficiency, which reminds me of the studies on copper deficiency which showed that the symptoms of copper-deficiency are worse if the animal is also consuming fructose. We have seen that hypers have increased symptoms after eating fruit and this effect may be due to fructose increasing copper-deficiency symptoms. It would be very interesting to know how this fructose effect works--perhaps not by increasing copper deficiency itself but because it works like copper-deficiency in increasing the magnesium deficiency effects.

Magnes Res 2000 Mar;13(1):19-27 t


Magnesium deficiency in the rat: effects of fructose, boron and copper.

Kenney MA, McCoy JH

School of Human Environmental Sciences, University of Arkansas, Fayetteville 72701, USA. kenney@comp.uark.edu

Magnesium (Mg) participates in many biochemical reactions which involve a variety of other nutrients. To elucidate some nutrient interactions, fructose (FR) and starch (ST) were compared as carbohydrate sources, and boron (B) and copper (Cu) were added to low-Mg diets for young male rats. Lack of Mg always caused characteristic deficiency symptoms. FR resembled Mg deficiency in effects on body, liver, and kidney weights and on plasma cholesterol level, but did not affect serum Mg or calcium (Ca). FR effects apparently were not mediated by changes in plasma Mg and Ca concentrations and were not prevented by adding Cu. Boron appeared to lessen effects of a low-Mg diet on body growth, serum cholesterol, and ash concentration in bone, but exacerbated deficiency symptoms, without affecting the concentration of Mg or Ca in serum. Results suggest that increased FR intake and marginal B might adversely affect individuals whose Mg status is suboptimal.

The following study, although enigmatic, appears to show that boron may affect thyroid function and the levels of T4 and T3.
Vopr Kurortol Fizioter Lech Fiz Kult 1989 May-Jun;(3):28-31


[Morphofunctional characteristics of the thyroid and a change in the level of thyroid hormones in the blood from the internal use of boron-containing waters].

[Article in Russian]

Korolev IuN, Panova LN, Bobkova AS, Korovkina EG

It has been established that intake of waters identical by Br concentrations (250 mg/l) but different by an ion-salt base leads to various structural changes of the thyroid at the tissue, cellular and subcellular levels. Artificial Br-containing water induces more pronounced shifts correlating with T3 and T4, blood concentrations. The ion-salt base was found essential in the mechanism of action of Br-containing water.

The following study shows that boron supplementation in males can increase estradiol (estrogen) and testosterone levels. This suggests that boron is involved in the conversion of progesterone into estradiol and testosterone. Since we have seen that hypers often have high progesterone levels and low estradiol levels (testosterone levels not known), this study offers more evidence that a boron deficiency may be involved in hyperthyroidism.
Biol Trace Elem Res 1997 Mar;56(3):273-86


The effect of boron supplementation on its urinary excretion and selected cardiovascular risk factors in healthy male subjects.

Naghii MR, Samman S

Department of Biochemistry, University of Sydney, NSW, Australia.

Boron (B) is an essential trace element for plants and its interrelationship with mineral and bone metabolism and endocrine function in humans has been proposed. Relatively little is known about the occurrence of B in the food chain and hence a biomarker which reflects its intake is required. Two studies were carried out to quantify the urinary B concentration of subjects consuming their habitual diet and the effect of supplementation. In addition, the effect of supplementation on plasma lipoprotein cholesterol concentrations and susceptibility to oxidation and plasma steroid hormones were determined. Boron excretion, obtained on two different occasions from 18 healthy male subjects, was found to be in the range 0.35-3.53 mg/day, with no significant difference between the two occasions. Supplementation with 10 mg B/d for 4 wk resulted in 84% of the supplemented dose being recovered in the urine. Plasma estradiol concentrations increased significantly as a result of supplementation (51.9 +/- 21.4 to 73.9 +/- 22.2 pmol/L; p < 0.004) and there was a trend for plasma testosterone levels to be increased. However, there was no difference in plasma lipids or the oxidizability of low-density lipoprotein. Our studies suggest that the absorption efficiency of B is very high and estimation of the urinary B concentration may provide a useful reflection of B intake. In addition, the elevation of endogenous estrogen as a result of supplementation suggests a protective role for B in atherosclerosis.

The following study indicates that boron is involved in cognitive performance. Because of the observed decreases in mental functioning in thyroid disease it's important to consider all nutrients which may be involved in brain function. Also note that boron may be involved in membrane function. Since boron is a light element and many of the lighter elements are involved in the passage of the heavier elements through the cell walls, boron's function may be involved in this process. We have seen that other light elements like lithium perform functions regulating the passage of heavier elements like copper into the cells.
Environ Health Perspect 1994 Nov;102 Suppl 7:65-72


Dietary boron, brain function, and cognitive performance.

Penland JG

United States Department of Agriculture, Agricultural Research Service, Grand Forks, North Dakota 58202-9034.

Although the trace element boron has yet to be recognized as an essential nutrient for humans, recent data from animal and human studies suggest that boron may be important for mineral metabolism and membrane function. To investigate further the functional role of boron, brain electrophysiology and cognitive performance were assessed in response to dietary manipulation of boron (approximately 0.25 versus approximately 3.25 mg boron/2000 kcal/day) in three studies with healthy older men and women. Within-subject designs were used to assess functional responses in all studies. Spectral analysis of electroencephalographic data showed effects of dietary boron in two of the three studies. When the low boron intake was compared to the high intake, there was a significant (p < 0.05) increase in the proportion of low-frequency activity, and a decrease in the proportion of higher-frequency activity, an effect often observed in response to general malnutrition and heavy metal toxicity. Performance (e.g., response time) on various cognitive and psychomotor tasks also showed an effect of dietary boron. When contrasted with the high boron intake, low dietary boron resulted in significantly poorer performance (p < 0.05) on tasks emphasizing manual dexterity (studies II and III); eye-hand coordination (study II); attention (all studies); perception (study III); encoding and short-term memory (all studies); and long-term memory (study I). Collectively, the data from these three studies indicate that boron may play a role in human brain function and cognitive performance, and provide additional evidence that boron is an essential nutrient for humans.

The following study is a gold mine. The study shows that boron supplementation increases estradiol and testosterone and for reasons given above I believe that these results suggest that boron might be deficient in hyperthyroidism. Additionally boron was shown to decrease plasma concentrations of calcium. High calcium levels may be associated with increased heart rate. Since calcium and magnesium act as antagonists, this reduction of calcium by boron may allow magnesium levels to rise and thereby lower the heart rate and muscle cramps.

Additionally boron was shown to increase plasma copper, copper-zinc superoxide dismutase (SOD is one of the body's most important free radical scavengers), and ceruloplasmin (a protein which transports copper). Here is direct evidence that boron is essential for copper metabolism and therefore quite probably for the correction of hyperthyroidism and possibly hypothyroidism.

Furthermore, the study offers a possible explanation for why estrogen may slow thyroid function: it increases plasma copper, SOD, and ceruloplasmin. Boron also increased these variables whether estrogen was administered or not.

This is excellent documentation to support my observations that boron was important in my recovery from hyperT.
Environ Health Perspect 1994 Nov;102 Suppl 7:59-63


Biochemical and physiologic consequences of boron deprivation in humans.

Nielsen FH

United States Department of Agriculture, Agricultural Research Service, Grand Forks, North Dakota 58202-9034.

Boron deprivation experiments with humans have yielded some persuasive findings for the hypothesis that boron is an essential nutrient. In the first nutritional study with humans involving boron, 12 postmenopausal women first were fed a diet that provided 0.25 mg boron/2000 kcal for 119 days, and then were fed the same diet with a boron supplement of 3 mg boron/day for 48 days. The boron supplementation reduced the total plasma concentration of calcium and the urinary excretions of calcium and magnesium, and elevated the serum concentrations of 17 beta-estradiol and testosterone. This study was followed by one in which five men over the age of 45, four postmenopausal women, and five postmenopausal women on estrogen therapy were fed a boron-low diet (0.23 mg/2000 kcal) for 63 days, then fed the same diet supplemented with 3 mg boron/day for 49 days. The diet was low in magnesium (115 mg/2000 kcal) and marginally adequate in copper (1.6 mg/2000 kcal) throughout the study. This experiment found higher erythrocyte superoxide dismutase, serum enzymatic ceruloplasmin, and plasma copper during boron repletion than boron depletion. The design of the most recent experiment was the same as the second study, except this time the diet was adequate in magnesium and copper. Estrogen therapy increased plasma copper and serum 17 beta-estradiol concentrations; the increases were depressed by boron deprivation. Estrogen ingestion also increased serum immunoreactive ceruloplasmin and erythrocyte superoxide dismutase; these variables also were higher during boron repletion than depletion for all subjects, not just those ingesting estrogen.

The following study indicates that boron and molybdenum affect estrogen metabolism and concludes that "It is possible that high dietary intakes of boron or molybdenum could regulate the rate of catabolism, or even the metabolic fate of the major estrogens."
J Inorg Biochem 1992 May 15;46(3):153-60 t


Borate and molybdate inhibition of catechol estrogen and pyrocatechol methylation by catechol-O-methyltransferase.

Beattie JH, Weersink E

Division of Biochemical Sciences, Rowett Research Institute, Bucksburn, Aberdeen, U.K.

The possibility that boron and molybdenum anions can influence sex steroid metabolism by forming complexes with catechol estrogens has been studied in vitro. The formation of 2-methoxyestrone (2-OHE1 2-Me) from 2-hydroxyestrone (2-OHE1) by catechol-O-methyltransferase (COMT) was followed by measuring the transfer of the radiolabeled methyl group from S-adenosylmethionine. In the presence of both sodium tetraborate and sodium molybdate using a phosphate buffer medium, the formation of 2-OHE1 2-Me decreased as the anion:2-OHE1 molar ratio was increased. However, the reverse effect was observed when using a tris buffer medium and further investigation showed that phosphate and sulphate also enhanced COMT activity in a tris buffer medium. Boric acid affinity medium, used as a substitute for borate salt, also showed a negative relationship with enzyme activity in a phosphate buffer medium, and inhibition of methylation was more marked than with the free anion. Erythrocytes contain appreciable amounts of COMT, which is mostly responsible for the rapid O-methylation of catechol estrogens in blood. The methylation of a simple catechol compound, 1,2-dihydroxybenzene (pyrocatechol) was therefore studied using rat red blood cell lysates. Methylation was inhibited in a concentration-related manner by borate, as found in the studies of 2-OHE1. It is possible that high dietary intakes of boron or molybdenum could regulate the rate of catabolism, or even the metabolic fate of the major estrogens.

This is the original USDA study which showed that boron supplementation increases estrogen and testosterone in postmenopausal women. The study also showed that boron "markedly reduced the urinary excretion of calcium and magnesium," interacts with magnesium metabolism, and the boron effects were not negated by a high intake of aluminum (1000 mg per day). It seems as though boron conserves magnesium and calcium, prevents the bone demineralization, and protects against osteoporosis.
FASEB J 1987 Nov;1(5):394-7 t


Effect of dietary boron on mineral, estrogen, and testosterone metabolism in postmenopausal women.

Nielsen FH, Hunt CD, Mullen LM, Hunt JR

United States Department of Agriculture, Grand Forks Human Nutrition Research Center, North Dakota 58202.

A study was done to examine the effects of aluminum, magnesium, and boron on major mineral metabolism in postmenopausal women. This communication describes some of the effects of dietary boron on 12 women between the ages of 48 and 82 housed in a metabolic unit. A boron supplement of 3 mg/day markedly affected several indices of mineral metabolism of seven women consuming a low-magnesium diet and five women consuming a diet adequate in magnesium; the women had consumed a conventional diet supplying about 0.25 mg boron/day for 119 days. Boron supplementation markedly reduced the urinary excretion of calcium and magnesium; the depression seemed more marked when dietary magnesium was low. Boron supplementation depressed the urinary excretion of phosphorus by the low-magnesium, but not by the adequate-magnesium, women. Boron supplementation markedly elevated the serum concentrations of 17 beta-estradiol and testosterone; the elevation seemed more marked when dietary magnesium was low. Neither high dietary aluminum (1000 mg/day) nor an interaction between boron and aluminum affected the variables presented. The findings suggest that supplementation of a low-boron diet with an amount of boron commonly found in diets high in fruits and vegetables induces changes in postmenopausal women consistent with the prevention of calcium loss and bone demineralization.
Additional studies on boron:

Boron is an essential nutrient for certain organisms, notably vascular plants and diatoms. Cyanobacteria require boron for formation of nitrogen-fixing heterocysts and boron may be beneficial to animals. Boron deficiency in plants produces manifold symptoms: many functions have been postulated. Deficiency symptoms first appear at growing points, within hours in root tips and within minutes or seconds in pollen tube tips, and are characterized by cell wall abnormalities. Boron-deficient tissues are brittle or fragile, while plants grown on high boron levels may have unusually flexible or resilient tissues. Borate forms cyclic diesters with appropriate diols or polyols. The most stable are formed with cis-diols on a furanoid ring. Two compounds have this structure physiologically: ribose in ribonucleotides and RNA, and apiose in the plant cell wall. Germanium can substitute for boron in carrot cell cultures. Both boron and germanium are localized primarily in the cell wall. We postulate that borate-apiofuranose ester cross-links are the auxin-sensitive acid-growth link in vascular plants, that the cyanobacterial heterocyst envelope depends on borate cross-linking of mannopyranose and/or galactopyranose residues in a polysaccharide-lipid environment, and that boron in diatoms forms ester cross-links in the polysaccharide cell wall matrix rather than boron-silicon interactions. Complexing of ribonucleotides is probably a factor in boron toxicity.boron--chemistry and biology.doc

Interest in boron as a naturally occurring trace element nutrient from the food supply is increasing. Mounting evidence suggests that boron is essential to human beings. This study explores the major food and beverage contributors of boron and estimates of daily boron intake from the American diet. Previous estimates in the literature of dietary boron consumption are based on limited foods and population segments. In this study we provide a more comprehensive assessment of boron consumption by the US population. A boron nutrient database of 1,944 individual foods was developed. These foods represent 95.3% by weight of all foods consumed in the US Department of Agriculture 1989-1991 Continuing Survey of Food Intakes by Individuals (1989-1991 CSFII). The Boron Nutrient Database (version 1.0) was then linked to the 3-day food records of 11,009 respondents to the 1989-1991 CSFII to generate the average daily boron intake for each person. The weighted 5th percentile, median, mean, and 95th percentile boron intakes, respectively, are 0.43, 1.02, 1.17 and 2.42 mg/day for men; 0.33, 0.83, 0.96 and 1.94 mg/day for women; and 0.40, 0.86, 1.01 and 2.18 mg/day for pregnant women. For vegetarian adults, these intakes are 0.46, 1.30, 1.47 and 2.74 mg/day for men and 0.33, 1.00, 1.29 and 4.18 mg/day for women. The top 2 boron contributors, coffee and milk, are low in boron, yet they make up 12% of the total boron intake by virtue of the volume consumed. Among the top 50 boron contributors, peanut butter, wine, raisins, peanuts, and other nuts are high in boron. As more data become available on daily boron requirements, the results of this study may be used to assess whether Americans' daily intake of boron is adequate.boron--daily intake in American diet.doc

Biol Trace Elem Res 1998 Winter;66(1-3):319-30


The justification for providing dietary guidance for the nutritional intake of boron.

Nielsen FH

United States Department of Agriculture, Agricultural Research Service, Grand Forks Human Nutrition Research Center, ND 58202-9034, USA.

Because a biochemical function has not been defined for boron (B), its nutritional essentiality has not been firmly established. Nonetheless, dietary guidance should be formulated for B, because it has demonstrated beneficial, if not essential, effects in both animals and humans. Intakes of B commonly found with diets abundant in fruits, vegetables, legumes, pulses, and nuts have effects construed to be beneficial in macromineral, energy, nitrogen, and reactive oxygen metabolism, in addition to enhancing the response to estrogen therapy and improving psychomotor skills and cognitive processes of attention and memory. Perhaps the best-documented beneficial effect of B is on calcium (Ca) metabolism or utilization, and thus, bone calcification and maintenance. The paradigm emerging for the provision of dietary guidance that includes consideration of the total health effects of a nutrient, not just the prevention of a deficiency disease, has resulted in dietary guidance for chromium (Cr) and fluoride; both of these elements have beneficial effects in humans, but neither has a defined biochemical function. Knowledge of B nutritional effects in humans equals or is superior to that of Cr and fluoride; thus, establishing a dietary reference intake for B is justified. An analysis of both human and animal data suggests that an acceptable safe range of population mean intakes of B for adults could well be 1-13 mg/d. Recent findings indicate that a significant number of people do not consistently consume more than 1 mg B/d; this suggests that B could be a practical nutritional or clinical concern.
Biol Trace Elem Res 1988 Sep-Dec;17:91-107


Magnesium and methionine deprivation affect the response of rats to boron deprivation.

Nielsen FH, Shuler TR, Zimmerman TJ, Uthus EO

US Department of Agriculture, Grand Forks Human Nutrition Research Center, ND 58202.

A series of nine experiments were done to obtain further evidence that boron might be involved in major mineral metabolism (Ca, P, and Mg), thus indicating that boron is an essential nutrient for animals. Eight factorially arranged experiments of 6-10 wk durations were done with weanling Sprague-Dawley male rats. One factorially arranged experiment was done with weanling spontaneously hypertensive rats. The variables in each experiment were dietary boron supplements of 0 and 3 micrograms g, and dietary magnesium supplements of either 200 (Experiments 1-3) or 100 (Experiments 4-9) and 400 micrograms/g. In Experiments 7 and 9, a third variable was dietary manganese supplements of 25 and 50 micrograms/g. Methionine status was varied throughout the series of experiments by supplementing the casein-based diet with methionine and arginine. Findings were obtained indicating that the severity of magnesium deprivation and the methionine status of the rat strongly influence the extent and nature of the interaction between magnesium and boron, and the response to boron deprivation. When magnesium deprivation was severe enough to cause typical signs of deficiency, a significant interaction between boron and magnesium was found. Generally, the interaction was characterized by the deprivation of one of the elements making the deficiency signs of the other more marked. The interaction was most evident when the diet was not supplemented with methionine and especially when the diet contained luxuriant arginine. Signs of boron deprivation were also more marked and consistent when the diet contained marginal methionine and luxuriant arginine. Among the signs of boron deprivation exhibited by rats fed marginal methionine were depressed growth and bone magnesium concentration, and elevated spleen wt/body wt and kidney wt/body wt ratios. Because the boron supplement of 3 micrograms/g did not make the dietary intake of this element unusual, it seems likely that the response of the rats to dietary boron in the present study were manifestations of physiological, not pharmacological, actions, and support the hypothesis that boron is an essential nutrient for the rat.

I believe that the boron you are taking will help with the yeast; and possibly also with other fermentation in your body; cancer cells typically ferment; they cannot live long in an oxygen rich environment.
Borax is perfect to combat both; as it has some H202 (oxygen) quality which is bad for the cancer cells, and the yeast cant survive high boron.

"ARTHRITIS IS A MINERAL DEFICIENCY - BORON IN PARTICULAR."

So claims Rex Newnham PhD DO ND, in his book "Away with Arthritis" (2nd edition printed 1993). Rex, a Western Australian now resident in the UK, lived at Guildford in the early 1960's. He taught chemistry, biology, geology, soil science and agricultural botany. In 1963 Rex moved to a house in a sandy area of Perth rather than the clay soil at Guildford. He had grown his own vegetables at both places but found that in sandy soil his crops showed signs of mineral deficiencies and he developed arthritis. With his scientific background Rex looked for the cause in recent changes. As an agriculturist he knew that boron was necessary for green plants, where it helps in calcium metabolism. In pursuing this he decided to trial it on himself using borax, a boron extract.
Rex took 30 mg of borax, twice a day. In 3 weeks the pain swelling and stiffness had gone. He stopped taking it . A year later the symptoms came back and disappeared quickly again on resumption of his borax dose. (older people may take longer)
Over the next 10 years he told many people he met and they tried borax with similar results. In 1976 Rex had some boron tablets made up. By the early 80's he was selling 10,000 bottles a month. Rex approached a drug company marketing a common painkilling drug for arthritis. They were only concerned about loss of their own profits and ended up instigating a government law that boron be declared a poison in any concentration. This resulted in Rex being fined and made boron unavailable. Boron is toxic if taken in large quantities. Rex points out that ordinary salt is toxic as well if too much is taken but salt is not banned.

Rex has since researched the effects of boron . It is well known that Carnarvon is a good place for arthritis. Many elderly people go there for a couple of months at a time to get rid of their arthritis. In 1981 Rex employed a number of school leavers to carry out a survey in Carnarvon. This showed that only 1% of the local population had arthritis. Other main Australian towns had a 20% rate of arthritis. Rex investigated further and found that it was not the climate that helped but the high boron content in water & food grown in the Gascoyne River bed.



Rex researched many areas of the world looking at arthritis incidence and soil content. Israel has some of the best soil in the world and a high boron content. An independent survey in 1978-1980 at the Kaplan Hospital showed arthritis for the whole country to be less than 1%.

In New Zealand a number of spas pools are recommended as being good for arthritis. They all have high boron readings. One motel at Ngawha has a collection of wheelchairs and crutches left behind by people no longer needing them when they leave.

The NZ green lipped mussel extract that is claimed to relieve arthritis was originally gathered from White Island, a volcanic island with vents under the sea. The mussels absorbed boron from the sea in this way and this was the secret to their success. Mussels gathered from other areas do not have the same effect as they lack the boron.

There are a number of degenerative diseases that appear to benefit from boron supplementation.

RHEUMATOID ARTHRITIS
- An autoimmune disease whereby the body attacks its own joints. The synovial membrane is affected causing degeneration and deformity of the joint. It is more common in women and often later develops into osteo arthritis. Work by a Prof Wyburn-Mason identified an amoeba as a parasite in the joint. Certain common minerals in the blood, including boron, can control these parasites.

OSTEO ARTHRITIS
- A wearing away of the joint, generally in joints that have done the most work or that have been damaged. The cartilage or tough fibrous matter around the joint wears away, then bone rubs on bone causing pain and permanent damage to the joint. Osteo arthritis occurs more often in men.

JUVENILE ARTHRITIS (Still's Disease)
- Similar to rheumatoid but occurs in young children. Can involve spleen, lymph and heart. On the increase. Often seen as 'growing pains' Responds well to certain minerals, like boron.

PYORRHOEA
- Loose teeth with swollen gums. It is really arthritis of the teeth and gums. Boron mouth-washes are recognised treatments available for pyorrhoea.

SPONDYLITIS
- Arthritis of the spine with inflammation in the joints. Calcium is lost from the vertebrae resulting in fusion and disc degeneration. Spondylitis also responds to mineral treatment like boron.

GOUT
- Again joint disease - often the big toe - where uric acid from the breakdown of proteins, concentrates in an area. The culprit again is a mineral imbalance from inefficient metabolism of sugars, alcohol and protein. Boron is needed to repair the damage.

OSTEOPOROSIS
- Decalcification of the bones. It is not a problem in primitive cultures. Osteoporosis is a disease of western civilisation due to the imbalance of bone minerals. As minerals are leached out for other body usage, the stability of bony tissue is eroded resulting in easy fracture. Crumbling limestone is a good example of osteoporosis.

Our bones are our mineral bank - the body's survival mechanism. Minerals are deposited and are the matrix and strength of the the bone. However if the there is a shortage of a particular mineral in another part of the body, a withdrawal will occur from the bone to provide for the deficit. After all, what use are bones if the body doesn't survive?

Bone is composed of many minerals in differing proportions - calcium, phosphorus, magnesium, sodium, sulphur, potassium, zinc, manganese, copper, silicon, stronium, boron. Boron helps other minerals to incorporate into the bony matrix. Body hormones and Vitamin D are also necessary. Bone is destroyed and replaced periodically just like leaves on a tree. Another ongoing function of bone is the manufacture of blood cells in the bone marrow. So we have a continuing need for the appropriate vitamins and minerals. We should be getting these from our diet. Again we can only get the minerals if they were in the soil in the first place. Plants and animals can display deficiencies too.
SIGNS of MINERAL DEFICIENCY in VEGETABLES
Leaves streaked with yellow,
Brassicas, eg cauliflower - hollow stems/browning
Potatoes - hollowed or browned insides
Sweet corn - aborted grains on cob
Apples - sunken dark spots under skin.
Grapes - aborted berries
Tomatoes - dark hollow areas on skin



Another contributor to arthritis is Fluoride. We have fluoride added to our water so we can't avoid it. Mottling of the teeth is a sign of too much fluoride. At the same time fluorosis of the skeletal bones occurs. Boron is the natural inactivator of fluoride.

Teeth are the only bones that are normally visible and their state is representative of other bones. If teeth are missing, filled or with cavities we must expect to have similar problems in other bones.

Sugar requires phosphorous for its metabolism as does white bread and caffeine (tea, coffee). Phosphorous is removed from the bone to metabolise these common foods where processing has removed many of the original minerals. Sugar utilises phosphorus, magnesium and chromium. When phosphorus is withdrawn from bone, calcium comes out too and as it is not needed is excreted in the urine. Soft drinks have both sugar and more phosphorus than is needed for sugar metabolism. The excess phosphorous combines with calcium upsetting the calcium phosphorus balance resulting in less available calcium. Caffeine also requires phosphorus and magnesium to metabolise. Sodium and potassium are lost at the same time.

Gristle in our diet contains essential nutrients to repair cartilage but we don't eat it any more. We feed it to our dogs. (I remember my grandad telling me to eat my gristle, that it was good for me.) NB It helps pulled muscles too.
Using Borax for arthritis is an old remedy. Since we made this information available I have had a number of older people tell me that their grandparents used to lick the end of their finger, dip it in the powder and lick off the Borax to rid them of their joint pain. Borax is in the washing powder section of the supermarket.


Boron is only available in WA on a doctor's script as Osteopartite with boron - a Nutrisearch product that also contains other necessary minerals. Your doctor can obtain more information from Nutrawest Pty Ltd.

CAUTIONS
Extra boron should only be taken if people display signs and symptoms of deficiency as outlined in the problems above. Excess boron can cause problems too. Conditions like ADHD, dyslexia, autism, asthma, allergies, cystic fibrosis, liver and oseophageal cancer can be triggered or made worse with excess boron. Boron increases oestrogen and calcium levels and lowers B6, zinc, blood glucose. Signs of toxicity are nausea, vomiting, lethagy, dermatitis, diarhoea. The antidote is extra B2. Cautions are part of a book "Boron, Phenols and Health" 1995 by Mary Duncan, a West Australian nurse.

Whip wrote:I agree, i have the terminator II zapper.

There could be a really good arthritis combo in taking internal boron, topical magnesium sprayed on the pain areas and a powerful internal bacteria/parasite killer.


Let me know what you guys think of this:

http://www.garynull.com/documents/Arthritis/free-living_amoeba.htm

"An interaction among B, Mg, and Mo affected plasma uric acid and glucose concentrations."

Prague wrote:no! boric acid is toxic - look for borax (or sodium borate if i remember well it in english)