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What does androgenetic alopecia have to do with COVID-19? An insight into a potential new therapy
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What does androgenetic alopecia have to do with COVID-19? An insight into a potential new therapy
Dermatol Ther. 2020 Jul;33(4):e13365. doi: 10.1111/dth.13365. Epub 2020 Apr 8.
What does androgenetic alopecia have to do with COVID-19? An insight into a potential new therapy
Andy Goren 1, John McCoy 1, Carlos G Wambier 2, Sergio Vano-Galvan 3, Jerry Shapiro 4, Rachita Dhurat 5, Kenneth Washenik 4 6, Torello Lotti 7
Affiliations expand
PMID: 32237190 PMCID: PMC7228378 DOI: 10.1111/dth.13365
Normally, I might be inclined not to post something like this, because of study authors flailing opinions on something
that has no basis in the scientific method. Nonetheless, the hypothesis here is quite relevant to real matters of MPB.
The full study link is below, however I will just paste and interject my own opinion on it as I've looked into this specific action for over the last 5 years in depth.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7228378/
If we cast aside the notion of "spike protein" and an unproven virus, and get into the actions of the
syndrome itself, it all leads to pathological iron overload.
The study authors inject absurd, unproven assertions repeatedly throughout this article, so here are the vital details that might be more meaningful.
TMPRSS2 expression is associated with an increase in androgen receptor (AR) expression.
Interestingly, angiotensin‐converting enzyme 2 (ACE2) has been shown to have reduced activity by the decrease of androgen hormones (experimental orchidectomy), possibly by decreased expression of ACE2.
TMPRSS2 gene transcription promoter site requires an activated androgen receptor, with androgens such as testosterone. Dihydrotestosterone (DHT) a potent androgen receptor activator and is intracellularly produced in particular cells of tissues such as prostate, hair, and liver that express 5‐alpha‐reductases, the targeted enzyme for drugs such as dutasteride and finasteride (5‐alpha‐reductase inhibitors).
Natural things that improve this situation:
Melatonin, Vitamin D, Quercetin, N-Acetylcysteine (NAC).
Really sad that the psychopathic medical cartel have already forced Amazon to strip NAC off the site, then talks of banning Vitamin D.
We can look at research here on what NAC does for the syndrome they call Certificate of Va((ine Identification (Spike-protein 19th letter of the alphabet).
I'll also point out that wireless (WiFi), EMF, 5th Generation Technology (short millimetre wavelengths) disrupt Vitamin D synthesis and Melatonin production.
What does androgenetic alopecia have to do with COVID-19? An insight into a potential new therapy
Andy Goren 1, John McCoy 1, Carlos G Wambier 2, Sergio Vano-Galvan 3, Jerry Shapiro 4, Rachita Dhurat 5, Kenneth Washenik 4 6, Torello Lotti 7
Affiliations expand
PMID: 32237190 PMCID: PMC7228378 DOI: 10.1111/dth.13365
Normally, I might be inclined not to post something like this, because of study authors flailing opinions on something
that has no basis in the scientific method. Nonetheless, the hypothesis here is quite relevant to real matters of MPB.
The full study link is below, however I will just paste and interject my own opinion on it as I've looked into this specific action for over the last 5 years in depth.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7228378/
If we cast aside the notion of "spike protein" and an unproven virus, and get into the actions of the
syndrome itself, it all leads to pathological iron overload.
The study authors inject absurd, unproven assertions repeatedly throughout this article, so here are the vital details that might be more meaningful.
TMPRSS2 expression is associated with an increase in androgen receptor (AR) expression.
Interestingly, angiotensin‐converting enzyme 2 (ACE2) has been shown to have reduced activity by the decrease of androgen hormones (experimental orchidectomy), possibly by decreased expression of ACE2.
TMPRSS2 gene transcription promoter site requires an activated androgen receptor, with androgens such as testosterone. Dihydrotestosterone (DHT) a potent androgen receptor activator and is intracellularly produced in particular cells of tissues such as prostate, hair, and liver that express 5‐alpha‐reductases, the targeted enzyme for drugs such as dutasteride and finasteride (5‐alpha‐reductase inhibitors).
Natural things that improve this situation:
Melatonin, Vitamin D, Quercetin, N-Acetylcysteine (NAC).
Really sad that the psychopathic medical cartel have already forced Amazon to strip NAC off the site, then talks of banning Vitamin D.
We can look at research here on what NAC does for the syndrome they call Certificate of Va((ine Identification (Spike-protein 19th letter of the alphabet).
I'll also point out that wireless (WiFi), EMF, 5th Generation Technology (short millimetre wavelengths) disrupt Vitamin D synthesis and Melatonin production.
_________________
My regimen
http://www.immortalhair.org/mpb-regimen
Now available for consultation (hair and/or health)
http://www.immortalhair.org/health-consultation
MikeGore likes this post
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