White Rice? Is it really that bad?

finemrespice wrote:white rice, is our main food, in korea. i am trying to reduce my rice diet these days. Diabetes are an issue in this country and it might actually be due to rice.

are you guys against white rice?

finemrespice wrote:white rice, is our main food, in korea. i am trying to reduce my rice diet these days. Diabetes are an issue in this country and it might actually be due to rice.

are you guys against white rice?

finemrespice wrote:but why do some people say to avoid white rice, especially those in the field of paleo-diet?

white rice are high in carbs, which will boost the sugar level in your blood which can turn to inflammation, no?

alot of koreans in my country are turning away from plain white rice to a mixture type. doctors also recommend to stay away from white rice if you;re a diabetic.

therefore, white rice (high in sugar) can not be optimal?

I suggest you look at the science more closely. It is not as simple as boosting blood sugar = inflammation. Asians have been eating white rice for centuries without diabetes epidemics.

BACKGROUND: Circulating levels of interleukin-6 (IL-6) and tumor necrosis factor-alpha (TNF-alpha) are elevated in diabetic patients. We assessed the role of glucose in the regulation of circulating levels of IL-6, TNF-alpha, and interleukin-18 (IL-18) in subjects with normal or impaired glucose tolerance (IGT), as well as the effect of the antioxidant glutathione.

METHODS AND RESULTS: Plasma glucose levels were acutely raised in 20 control and 15 IGT subjects and maintained at 15 mmol/L for 5 hours while endogenous insulin secretion was blocked with octreotide. In control subjects, plasma IL-6, TNF-alpha, and IL-18 levels rose (P<0.01) within 2 hours of the clamp and returned to basal values at 3 hours. In another study, the same subjects received 3 consecutive pulses of intravenous glucose (0.33 g/kg) separated by a 2-hour interval. Plasma cytokine levels obtained at 3, 4, and 5 hours were higher (P<0.05) than the corresponding values obtained during the clamp. The IGT subjects had fasting plasma IL-6 and TNF-alpha levels higher (P<0.05) than those of control subjects. The increase in plasma cytokine levels during the clamping lasted longer (4 hours versus 2 hours, P<0.01) in the IGT subjects than in the control subjects, and the cytokine peaks of IGT subjects after the first glucose pulse were higher (P<0.05) than those of control subjects. On another occasion, 10 control and 8 IGT subjects received the same glucose pulses as above during an infusion of glutathione; plasma cytokine levels did not show any significant change from baseline after the 3 glucose pulses.

CONCLUSIONS: Hyperglycemia acutely increases circulating cytokine concentrations by an oxidative mechanism, and this effect is more pronounced in subjects with IGT. This suggests a causal role for hyperglycemia in the immune activation of diabetes.

CausticSymmetry wrote:The question defined, is eating rice bad for hair? Not if simple precautions are taken.

mphatesmpb wrote:

I suggest you look at the science more closely. It is not as simple as boosting blood sugar = inflammation. Asians have been eating white rice for centuries without diabetes epidemics.

That's not true. Hyperglycemia does cause systemic inflammation. Look at this abstract:

BACKGROUND: Circulating levels of interleukin-6 (IL-6) and tumor necrosis factor-alpha (TNF-alpha) are elevated in diabetic patients. We assessed the role of glucose in the regulation of circulating levels of IL-6, TNF-alpha, and interleukin-18 (IL-18) in subjects with normal or impaired glucose tolerance (IGT), as well as the effect of the antioxidant glutathione.

METHODS AND RESULTS: Plasma glucose levels were acutely raised in 20 control and 15 IGT subjects and maintained at 15 mmol/L for 5 hours while endogenous insulin secretion was blocked with octreotide. In control subjects, plasma IL-6, TNF-alpha, and IL-18 levels rose (P<0.01) within 2 hours of the clamp and returned to basal values at 3 hours. In another study, the same subjects received 3 consecutive pulses of intravenous glucose (0.33 g/kg) separated by a 2-hour interval. Plasma cytokine levels obtained at 3, 4, and 5 hours were higher (P<0.05) than the corresponding values obtained during the clamp. The IGT subjects had fasting plasma IL-6 and TNF-alpha levels higher (P<0.05) than those of control subjects. The increase in plasma cytokine levels during the clamping lasted longer (4 hours versus 2 hours, P<0.01) in the IGT subjects than in the control subjects, and the cytokine peaks of IGT subjects after the first glucose pulse were higher (P<0.05) than those of control subjects. On another occasion, 10 control and 8 IGT subjects received the same glucose pulses as above during an infusion of glutathione; plasma cytokine levels did not show any significant change from baseline after the 3 glucose pulses.

CONCLUSIONS: Hyperglycemia acutely increases circulating cytokine concentrations by an oxidative mechanism, and this effect is more pronounced in subjects with IGT. This suggests a causal role for hyperglycemia in the immune activation of diabetes.

This is not directed at abc123, but I've noticed a ridiculous pattern on this forum:
1. CS says x.
2. Forum participant disagrees with x, and presents valid reasoning (scientific studies)
3. Other forum participants clowns on the person disagreeing with CS, telling him to "look at the science more carefully," as if they thoroughly understand the science themselves. Usually the most sophisticated reasoning backing their opinion is something like "well...Asians (or Eskimos, Kitavans...whatever) have been doing it for thousands of years, and they seem to be fine." That's bullshit.

I understand that CS knows a lot about this stuff, but he doesn't know everything. This is a forum for discussion and disagreement, not for blindly agreeing with whatever CS says.

mphatesmpb wrote:

I suggest you look at the science more closely. It is not as simple as boosting blood sugar = inflammation. Asians have been eating white rice for centuries without diabetes epidemics.

That's not true. Hyperglycemia does cause systemic inflammation. Look at this abstract:

BACKGROUND: Circulating levels of interleukin-6 (IL-6) and tumor necrosis factor-alpha (TNF-alpha) are elevated in diabetic patients. We assessed the role of glucose in the regulation of circulating levels of IL-6, TNF-alpha, and interleukin-18 (IL-18) in subjects with normal or impaired glucose tolerance (IGT), as well as the effect of the antioxidant glutathione.

METHODS AND RESULTS: Plasma glucose levels were acutely raised in 20 control and 15 IGT subjects and maintained at 15 mmol/L for 5 hours while endogenous insulin secretion was blocked with octreotide. In control subjects, plasma IL-6, TNF-alpha, and IL-18 levels rose (P<0.01) within 2 hours of the clamp and returned to basal values at 3 hours. In another study, the same subjects received 3 consecutive pulses of intravenous glucose (0.33 g/kg) separated by a 2-hour interval. Plasma cytokine levels obtained at 3, 4, and 5 hours were higher (P<0.05) than the corresponding values obtained during the clamp. The IGT subjects had fasting plasma IL-6 and TNF-alpha levels higher (P<0.05) than those of control subjects. The increase in plasma cytokine levels during the clamping lasted longer (4 hours versus 2 hours, P<0.01) in the IGT subjects than in the control subjects, and the cytokine peaks of IGT subjects after the first glucose pulse were higher (P<0.05) than those of control subjects. On another occasion, 10 control and 8 IGT subjects received the same glucose pulses as above during an infusion of glutathione; plasma cytokine levels did not show any significant change from baseline after the 3 glucose pulses.

CONCLUSIONS: Hyperglycemia acutely increases circulating cytokine concentrations by an oxidative mechanism, and this effect is more pronounced in subjects with IGT. This suggests a causal role for hyperglycemia in the immune activation of diabetes.

This is not directed at abc123, but I've noticed a ridiculous pattern on this forum:
1. CS says x.
2. Forum participant disagrees with x, and presents valid reasoning (scientific studies)
3. Other forum participants clowns on the person disagreeing with CS, telling him to "look at the science more carefully," as if they thoroughly understand the science themselves. Usually the most sophisticated reasoning backing their opinion is something like "well...Asians (or Eskimos, Kitavans...whatever) have been doing it for thousands of years, and they seem to be fine." That's bullshit.

I understand that CS knows a lot about this stuff, but he doesn't know everything. This is a forum for discussion and disagreement, not for blindly agreeing with whatever CS says.

finemrespice wrote:but why do some people say to avoid white rice, especially those in the field of paleo-diet?

white rice are high in carbs, which will boost the sugar level in your blood which can turn to inflammation, no?

alot of koreans in my country are turning away from plain white rice to a mixture type. doctors also recommend to stay away from white rice if you;re a diabetic.

therefore, white rice (high in sugar) can not be optimal?