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Iodine hair-loss ; 4 days on / 4 days off with Supplements/vitamins....
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Iodine hair-loss ; 4 days on / 4 days off with Supplements/vitamins....
CS and others...
this is very interesting information about iodine and hair loss/regrowth...
ted talks about taking iodine and supplements/vitamins....he suggests 4 days on...4 days off to allow the body to detox. Is this accurate? i read other places that 6 days on 1 day off..
what is the best way here?
http://www.earthclinic.com/CURES/hair_loss.html
this is very interesting information about iodine and hair loss/regrowth...
ted talks about taking iodine and supplements/vitamins....he suggests 4 days on...4 days off to allow the body to detox. Is this accurate? i read other places that 6 days on 1 day off..
what is the best way here?
http://www.earthclinic.com/CURES/hair_loss.html
RobHealthMan- Posts : 451
Join date : 2009-11-07
Re: Iodine hair-loss ; 4 days on / 4 days off with Supplements/vitamins....
RobHealthMan - That's a rather impressive write up about hair loss.
I'm not absolutely certain about the breaks, because most of the research I draw off comes from Dr. Abraham and Dr. David Brownstein, who in my opinion are ranked 2nd to none in clinical and iodine research in the world.
In any event, Ted's mention of mycoplasma is interesting and it has receive very little attention on this forum, and surprisingly so. It is a classical indicator of rheumatoid arthritis for example. A condition that has some similarities to MPB pathology.
The fungus he discusses is most likely species of malassezia (yeasts) and candida. However, the role of bacteria is probably the most important, because getting rid of the yeasts does not address entirely the problem. However, having fungus is a consequence of poor tissue oxygenation--which my view and research is at the root core of hair loss.
Poor oxygen respiration can allow something like a fungus to thrive.
Here is a recent study I will be posting:
Free Radic Biol Med. 2010 Mar 15;48(6):781-90.
Impairment of mitochondrial respiratory chain activity in aortic endothelial cells induced by glycated low-density lipoprotein.
Sangle GV, Chowdhury SK, Xie X, Stelmack GL, Halayko AJ, Shen GX.
Department of Physiology, University of Manitoba, Winnipeg, Canada MB R3E 3P4.
Coronary artery disease (CAD) is the leading cause of mortality in diabetic patients. Mitochondrial dysfunction and increased production of reactive oxygen species (ROS) are associated with diabetes and CAD. Elevated levels of glycated LDL (glyLDL) were detected in patients with diabetes. Our previous studies demonstrated that glyLDL increased the generation of ROS and altered the activities of antioxidant enzymes in vascular endothelial cells (EC). This study examined the effects of glyLDL on oxygen consumption in mitochondria and the activities of key enzymes in the mitochondrial electron transport chain (ETC) in cultured porcine aortic EC. The results demonstrated that glyLDL treatment significantly impaired oxygen consumption in Complexes I, II/III, and IV of the mitochondrial ETC in EC compared to LDL or vehicle control detected using oxygraphy. Incubation with glyLDL significantly reduced the mitochondrial membrane potential, the NAD(+)/NADH ratio, and the activities of mitochondrial ETC enzymes (NADH-ubiquinone dehydrogenase, succinate cytochrome c reductase, ubiquinone cytochrome c reductase, and cytochrome c oxidase) in EC compared to LDL or control. The abundance of mitochondria-associated ROS and the release of ROS from EC were significantly increased after glyLDL treatment. The findings suggest that glyLDL attenuates the activities of key enzymes in the mitochondrial ETC, decreases mitochondrial oxygen consumption, reduces mitochondrial membrane potential, and increases ROS generation in EC, which potentially contribute to mitochondrial dysfunction in diabetic patients.
Of course, sugars (which slow oxygen respiration) are a recipe for rheumatic conditions, yeasts and fungus.
I'm not absolutely certain about the breaks, because most of the research I draw off comes from Dr. Abraham and Dr. David Brownstein, who in my opinion are ranked 2nd to none in clinical and iodine research in the world.
In any event, Ted's mention of mycoplasma is interesting and it has receive very little attention on this forum, and surprisingly so. It is a classical indicator of rheumatoid arthritis for example. A condition that has some similarities to MPB pathology.
The fungus he discusses is most likely species of malassezia (yeasts) and candida. However, the role of bacteria is probably the most important, because getting rid of the yeasts does not address entirely the problem. However, having fungus is a consequence of poor tissue oxygenation--which my view and research is at the root core of hair loss.
Poor oxygen respiration can allow something like a fungus to thrive.
Here is a recent study I will be posting:
Free Radic Biol Med. 2010 Mar 15;48(6):781-90.
Impairment of mitochondrial respiratory chain activity in aortic endothelial cells induced by glycated low-density lipoprotein.
Sangle GV, Chowdhury SK, Xie X, Stelmack GL, Halayko AJ, Shen GX.
Department of Physiology, University of Manitoba, Winnipeg, Canada MB R3E 3P4.
Coronary artery disease (CAD) is the leading cause of mortality in diabetic patients. Mitochondrial dysfunction and increased production of reactive oxygen species (ROS) are associated with diabetes and CAD. Elevated levels of glycated LDL (glyLDL) were detected in patients with diabetes. Our previous studies demonstrated that glyLDL increased the generation of ROS and altered the activities of antioxidant enzymes in vascular endothelial cells (EC). This study examined the effects of glyLDL on oxygen consumption in mitochondria and the activities of key enzymes in the mitochondrial electron transport chain (ETC) in cultured porcine aortic EC. The results demonstrated that glyLDL treatment significantly impaired oxygen consumption in Complexes I, II/III, and IV of the mitochondrial ETC in EC compared to LDL or vehicle control detected using oxygraphy. Incubation with glyLDL significantly reduced the mitochondrial membrane potential, the NAD(+)/NADH ratio, and the activities of mitochondrial ETC enzymes (NADH-ubiquinone dehydrogenase, succinate cytochrome c reductase, ubiquinone cytochrome c reductase, and cytochrome c oxidase) in EC compared to LDL or control. The abundance of mitochondria-associated ROS and the release of ROS from EC were significantly increased after glyLDL treatment. The findings suggest that glyLDL attenuates the activities of key enzymes in the mitochondrial ETC, decreases mitochondrial oxygen consumption, reduces mitochondrial membrane potential, and increases ROS generation in EC, which potentially contribute to mitochondrial dysfunction in diabetic patients.
Of course, sugars (which slow oxygen respiration) are a recipe for rheumatic conditions, yeasts and fungus.
_________________
My regimen
http://www.immortalhair.org/mpb-regimen
Now available for consultation (hair and/or health)
http://www.immortalhair.org/health-consultation
Re: Iodine hair-loss ; 4 days on / 4 days off with Supplements/vitamins....
Would eating dulse provide the same benefits as supplementing lugos?
big mike- Posts : 120
Join date : 2009-09-26
Re: Iodine hair-loss ; 4 days on / 4 days off with Supplements/vitamins....
big mike - Probably not too much.
_________________
My regimen
http://www.immortalhair.org/mpb-regimen
Now available for consultation (hair and/or health)
http://www.immortalhair.org/health-consultation
Re: Iodine hair-loss ; 4 days on / 4 days off with Supplements/vitamins....
Thanks CS.
from your experience what is the best way to get rid of bacteria/fungus that attacks follicle? is it iodine internally and iodine topically?
probiotics? yogurt? these help?
thanks again!
from your experience what is the best way to get rid of bacteria/fungus that attacks follicle? is it iodine internally and iodine topically?
probiotics? yogurt? these help?
thanks again!
RobHealthMan- Posts : 451
Join date : 2009-11-07
Re: Iodine hair-loss ; 4 days on / 4 days off with Supplements/vitamins....
RobHealthMan - The short answer is to increase oxygen to tissue. Fungus, molds (cancer), yeast all grow in oxygen deprived environments. Optimizing thyroid function, through iodine and many, many other measures--all which indirectly benefit the thyroid and tissue oxygenation.
Removing toxic metals is quite important, inhibiting antiplasmin (improves coagulation balance), it goes on to the rest of a primary regimen.
Removing toxic metals is quite important, inhibiting antiplasmin (improves coagulation balance), it goes on to the rest of a primary regimen.
_________________
My regimen
http://www.immortalhair.org/mpb-regimen
Now available for consultation (hair and/or health)
http://www.immortalhair.org/health-consultation
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