D3

jdp701 wrote:
Anyhow, there is another thread, a more recent thread, at imminist showing an increase in certain cancers with D3 supplementation or increased 25 D levels... I forget which.

The Natural wrote:
JDP - Since you have decided to leave them unanswered, my questions now become rhetorical ones: You are not a professional trained in the areas of medicine or science; You are a layperson with Internet access, cherry picking ideas from Trevor Marshall.

here is a good link http://autoimmunityresearch.org/transcripts/waterhouse_lax2006.pdf

A bit more complicated than that. Oral administration of D is all unconverted and assumes that your receptors and conversion facilities work.
Because if they don't it works like an allergy pill that lowers your immune reaction to invading pathogens…

So the first set of “ra ra” news to push huge doses was shrinking of some tumors and less dead garbage in the blood. The question no one asked, and is stirred away from, is why? Did it cure cancer? Did it kill the pathogens?

What we find out now is that most people can't convert the oral D at all and it simply builds up lowering immunity.
Your best bet if unsure is still to get it from the sun since you body can and will turn off production of D. But some have gone beyond a mere build up and need to be completely removed from any D until better. These are people with underlying Lyme, which is also attempting to do what D does and that is mask any disease, that carry Rickettsia and or Sarcocystis which both produce\extract retro viruses that block these receptors.

As in the story of the person that had Ascaris and was pushed into taking Allergy pills by his well educated, well documented quack…those just made everything worse by both taxing the liver and stopping normal immune/inflammatory reactions.

I was well into the study of D by the time my News program was paying “specialists” to push multi thousand IU doses of D. This is because it red flagged in my Dad during bio-feedback and I actually watched his skin color change while using standard RSMF rife code on him.

researchers at the University of Science and Technology in Norway just released the results of a study that measured the forearm bone mineral density of 3,042 Norwegian women, aged 50 – 70 years old. They found that those women who had not taken cod liver oil (a substance that contains high levels of vitamin D) during childhood had higher bone mineral density compared to those who had ingested cod liver oil.[79] Since the study compared childhood intake of vitamin D to bone density at least 4-5 decades after ingestion, it is a good example of how only those studies which track vitamin D intake over long periods of time are likely to pick up on the harm the secosteroid causes in the longterm

Marshall discovered that once inside a cell, CWD bacteria activate an inflammatory pathway that is responsible for generating many of the painful symptoms of Th1 diseases. One of the major molecules involved in this pathway is 1,25D, an activated form of Vitamin D that functions as a hormone. Once inside a cell, CWD bacteria convert the regular form of Vitamin D (termed 25D) into 1,25D at a much higher rate than in healthy cells. Thus blood tests reveal that patients with Th1 diseases tend to display much higher levels of 1,25D than their healthy counterparts

1. Vitamin D is not a vitamin; it is an immunosuppressive steroid.
Let’s start with this fact: the vast majority of doctors touting the benefits of vitamin D are not aware of discoveries made by researchers in the field of molecular biology, which have clearly shown that the “vitamin” D derived from diet and supplements is not a vitamin, but a steroid with immunosuppressive properties when elevated.2
There are several forms of vitamin D. The form of vitamin D we get from food, diet, supplements and sun exposure is called D3. D3 is converted by the kidneys into 25-D, which functions as a steroid. 1,25-D, the activated form of vitamin D, functions as both a steroid and a hormone. It is produced inside various types of cells, including those of the immune system and the kidneys, as well as in response to sunlight. In healthy individuals, the kidneys continually convert 25-D into its active form, 1,25-D.34
According to a paper published by the Institute of Biomedical Research in Birmingham, England, “The active form of vitamin D, [1,25-D] is a potent immunomodulatory seco-steroid” meaning that it is a steroid-like molecule which is able to control the activity of the immune system.”5
Molecular modeling has shown that the hormonal 1,25-D form binds and activates the Vitamin D Receptor. The Vitamin D Receptor plays a fundamental role in the body. It transcribes 913 genes, and researchers at McGill University in Canada just released a paper saying it may actually transcribe 27,091.6 But, the Vitamin D Receptor also performs another critical function – it serves as a switch that regulates the activity of the innate immune system.789
A molecular model comparing the structure of 25-D and of 1,25-D10According to recent molecular models, the steroid 25-D binds the Vitamin D Receptor and affects the activity of the immune system as well, but in a manner opposite to 1,25-D. When the steroid 25-D binds the Vitamin D Receptor, it decreases the activity of the receptor, causing the innate immune system to slow down and shut off. This effect begins around 20 ng/ml and gradually increases with higher levels of 25-D, until the VDR becomes completely blocked.11
At the moment, most researchers understand that 1,25-D activates the Vitamin D Receptor. However, they are unaware of the models which demonstrate that 25-D has the opposite effect. Consequently, they do not understand that when people start to supplement with extra vitamin D (which is converted into 25-D) the Vitamin D Receptor begins to turn off, not on.
Most of these researchers are also unaware of a new understanding about the cause of many chronic diseases. As a person falls ill with a chronic disease, L-form bacteria begin to live inside the cells of the immune system and in various tissues.1213 These bacteria create proteins that, just like elevated 25-D, are able to bind and block the Vitamin D Receptor.14 Together, elevated 25-D and bacterial proteins block the ability of the Vitamin D Receptor to turn on the immune system more than either substance alone.

Introduction of oral vitamin D supplementation and the rise of the allergy pandemic

http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2794851/?tool=pmcentrez

Silica instead of Vitamin D in Autoimmune Diseases

http://www.ndaccess.com/EatingAlive/Page_Detail.asp?PageID=22&CommentID=15

Additive immunosuppressive effects of 1,25-dihydroxyvitamin D3 and corticosteroids on TH1, but not TH2, responses

http://www.ncbi.nlm.nih.gov/pubmed/11080724?ordinalpos=1&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_RVDocSum